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Disable Facebook Login Button after login

I’m working with a CRM application, it’s built using Drupal 7. I have a facebook login field on my user registration form, and when a user clicks the login button the first time, it logs the user in with a normal login/pass query.
The problem is that I would like the user to be able to save the form and login again through facebook once they have logged in, however I would like to disable the login button, they could then log in with normal login process by clicking the facebook login button again.
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Asigning the form to always be submitted/refreshed
On login

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I have a weird problem. Now, on this computer the activation date is 7/21/2013 and the license is good for 5 years! However on this new one the activation date is 4/11/2017 and the license is good for 3 years so why is there that difference? Is this normal?

The 4-digit activation codes are valid for 90 days, then they expire. After 90 days you must purchase a new code. The license expiration check happens when you open your software (unless you already have an old license).
In this case the new license is a bit more expensive to buy, but you can still use the software for the first three years.
You can check if the activation is in the past by choosing Help > About. On that screen, on the Activation tab, you’ll find a column labelled “Actual activation date”, which shows how long the license is valid for.

Siamese mice are excellent animal models for cancer biology and immunology.
Strategies for treating cancer are directed toward slowing the growth and spread of primary tumor cells and eliminating metastatic disease. However, there are many clinically important problems in the management of cancer that are not yet fully understood. A major challenge in cancer immunotherapy is to target immunologically inert or nonfunctional tumor cells. Selective immunomodulation of nonfunctional or apparently inert tumor cells may provide an alternative approach to cancer therapy. Because it is possible to select immunologically functional lymphocytes from tumor-bearing animals, but not immunologically inert T cells (i.e., T cells from tumor-bearing mice do not proliferate in response to superantigen), the development of models for T cell-mediated tumor protection is of major importance. We have shown that, by selecting appropriately, tumor-bearing animals exhibit changes in immune function that are identical to those of allogeneic (non-self) tumor-bearing animals. In particular, memory T cells from tumor-bearing mice are functional and bear a multitude of TCR specificity. With the discovery of the rearranged TCR repertoire in tumors, we now have a rational approach for selecting these specificities for antitumor therapy. The ease of tumor induction and the rapidity of onset of experimental tumors in the murine model make this an ideal model system for the study of tumor immunity and the development of new immunotherapies.Friedrich Eckstein

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