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Oral submucous fibrosis (OSMF) is a premalignant condition with an insidious onset and progression, and for this reason, it is called “The Great Pretender.”\[[@ref1]\] The disease is common in North India, although increasingly, Western countries are reporting an increase in incidence.\[[@ref2]\] The pathological origin of the disease is inflammatory reaction of the submucosa in response to chronic irritation due to increased oral habits and mainly tobacco chewing. The lesions affect the buccal mucosa and often extend into the surrounding oral cavity affecting the tongue and soft palate.\[[@ref3]\] The precise etiopathogenesis of OSMF remains unknown. Genetic factors, environmental factors, and nutritional modifications have been implicated in the pathogenesis of OSMF.\[[@ref4]\] The main pathological features include hyalosis of the fibrous tissue, overproduction of fibroblasts and collagen, and excessive deposition of glycoproteins.\[[@ref5]\]
Previous studies have established the beneficial effect of anti-tobacco interventions, such as lowering frequency and duration of tobacco chewing; elimination of smokeless tobacco; elimination of smoking; and reducing the number of cigarettes smoked by the patient.\[[@ref6]\] The medications that have been administered to OSMF patients include elimination or reduction of oral habits, antioxidant supplementation, corticosteroids, and surgical removal of excess fibrous tissue.\[[@ref7]\] There is a growing interest in dietary modifications, particularly in the use of fruits and vegetables.\[[@ref8]\]
India is a country with population of 1.25 billion. According to the 2011 World Health Organization report, 90% of the population in India is in rural areas and are mainly dependent on traditional medicine as a primary healthcare modality.\[[@ref9]\] The use of herbal or medicinal products has been a major source of healthcare for the rural population in India.\[[@ref10]\] Ayurveda is the major system of traditional medicine in India. The herbs used in Ayur
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N-Acetylneuraminic acid (9-O-acetyl-sialic acid) is present on the surface of many animal and plant cells. The recently described polyclonal antisera against N-acetylneuraminic acid, based on the keyhole limpet haemocyanin (KLH)-antigen, cross-react with the herpesvirus-like antigen of pig kidney cells. We have therefore used antisera of this type to examine herpes-virus-like infections in cultures of pig kidney cells. Cultured cells showed an increase in antigenicity, which was enhanced after an infection with pseudorabies virus. The increase was also seen after an infection with bovine herpesvirus-1, human herpesvirus-6 and human herpesvirus-7. These antisera also cross-react with the virus-like antigen of Marek’s disease virus and with the herpes
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